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Namely, during the prodromal stage of HD in humans, CAU shows significant and distinct alterations in gene expression that include upregulation of GSTO2 compared with the non-significant difference in controls (Fig

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Such neuronal changes, or lesions, are characterized by tangles of nerve fibers and the increased presence of -amyloid and hyperphosphorylated tau in the brain, which ultimately leads to a decline in nervous system function